Source: UNIVERSITY OF CALIFORNIA, DAVIS submitted to NRP
METABOLIC RELATIONSHIPS IN SUPPLY OF NUTRIENTS FOR LACTATING COWS
Sponsoring Institution
National Institute of Food and Agriculture
Project Status
COMPLETE
Funding Source
Reporting Frequency
Annual
Accession No.
1025816
Grant No.
(N/A)
Cumulative Award Amt.
(N/A)
Proposal No.
(N/A)
Multistate No.
NC-_old2040
Project Start Date
Feb 9, 2021
Project End Date
Jun 30, 2021
Grant Year
(N/A)
Program Code
[(N/A)]- (N/A)
Recipient Organization
UNIVERSITY OF CALIFORNIA, DAVIS
410 MRAK HALL
DAVIS,CA 95616-8671
Performing Department
Population Health & Reproduction
Non Technical Summary
Current practice in the California dairy industry is to inject an antibiotic into the teat when cows are dried off at the end of lactation. This practice is thought to aid the cow in treating bacterial infections and to prevent mastitis. But, treatment may reduce the efficacy of white blood cells by impairing mitochondrial function in peripheral blood mononuclear cells (PBMC) or somatic cells (SC) in the mammary gland. California Dairy Research Foundation (CDRF) has already funded the project exploring the impact of antibiotics on SC mitochondrial function in the mammary gland. This project addresses the extent to which mitochondrial function may be compromised in circulating PBMC by antibiotic use at the cessation of milking in the mammary gland. Approximately 20 cows per dairy (5 cows with low SCC (< 100,000) and no Ceftiofur, and 5 cows with low SCC and Ceftiofur; 5 cows with high SCC (> 200,000) and no Ceftiofur and 5 cows with high SCC and Ceftiofur) will be selected from 5 dairies to examine changes in circulating PBMC mitochondrial activity with antibiotic treatment. Dairies will treat 2 quarters per cow at dry off using Ceftiofur (Spectramast DC). Ceftiofur has been shown to change mitochondrial structure and reduce mitochondrial activity in white blood cells. Blood samples will be collected just prior to antibiotic treatment, 7 days later and finally 40 days in milk in the next lactation. Mitochondria will be isolated from PBMC to measure citrate synthase, and complexes I, IV and V activities. A reduced mitochondrial enzyme activity will indicate the effect of antibiotic treatment on mitochondrial PBMC activity. The results of this study will provide information supporting reduced use of antibiotics at cessation of milking and determine if there is a link between Ceftiofur use, mammary SC mitochondrial function, mastitis infection and transition disease.
Animal Health Component
20%
Research Effort Categories
Basic
60%
Applied
20%
Developmental
20%
Classification

Knowledge Area (KA)Subject of Investigation (SOI)Field of Science (FOS)Percent
30534101030100%
Knowledge Area
305 - Animal Physiological Processes;

Subject Of Investigation
3410 - Dairy cattle, live animal;

Field Of Science
1030 - Cellular biology;
Goals / Objectives
Objective 1: To quantify factors that impact supply and availability of nutrients utilized for efficient milk production while reducing environmental impact
Project Methods
The hypothesis is that Ceftiofur applied when cows are dried-off decreases PBMC mitochondrial function systemically as indicated by reduced mitochondrial citrate synthase, complex I, IV and V activities in PBMC and may increase transition issues. Cows (20 per dairy; 5 per treatment and SCC level) will be selected from 5 dairies to determine changes in mitochondrial activity of circulating PBMC due to antibiotic treatment with Ceftiofur at dry off. Cows will be enrolled at dry off according to SCC as determined by California Mastitis Test and milk samples will be taken to DHIA at each time point to quantify SCC in each quarter. Blood samples will be collected just prior to treatment at dry off, 7 days later and then 50 days after the next lactation begins. Mitochondria in PBMC will be isolated by membrane disruption and centrifugation. Mitochondrial enzyme activity (citrate synthase and complexes I, IV and V) will be assayed by colorimetric changes in a spectrophotometer and expressed relative to protein content (mOD/min/ug mitochondrial protein). Occurrence of transition diseases at 7 days in milk and from dairy herd management records. Enzyme activities in SC and circulating PBMC will be compared at each time point to determine the impact of Ceftiofur and SCC on mitochondrial function of immune cells. Data does not currently exist to estimate sample size. However, our past studies have shown that 5 cows are adequate to detect differences in PBMC mitochondrial enzyme activities. Differences in mitochondrial enzyme activities by treatment will be determined statistically using a mixed model with random effect of dairy. Occurrences of transition diseases will be compared across treatments with a similar mixed model.

Progress 02/09/21 to 06/30/21

Outputs
Target Audience:Target audience for this project is veterinarians, nutritionists, dairy owners and managers, Farm Advisors and other researchers in udder health. Changes/Problems: Nothing Reported What opportunities for training and professional development has the project provided?The project provided funding for 1 masters student and 1 phd student to complete theirresearch for their thesi.There were also 4 other graduate students that helped with this trial and gained valuable cow sampling and handling skills and learned more about the dairy industry since samples were obtained from 5 commercial dairies. How have the results been disseminated to communities of interest?The MS thesis has been submitted as degreee requirements and the first manuscript is currently under review. What do you plan to do during the next reporting period to accomplish the goals? Nothing Reported

Impacts
What was accomplished under these goals? Mitovchondria were isolated from milk and blood to determined their electron transport enzyme activities and how that would affect ATP generation at different times during the transition period and into the next lactation. These measurements also tell us if mitochondria metabolism in somatic cells or blood would be affected by antibiotic treatment (ie decreased ATP and therefore energy yeilds). Mitochondria are throught to be evolutionarily similar to bacteria and previous evidence in humans has shown that some antibiotics do impact mitochondiral metabolism.

Publications