Source: TENNESSEE STATE UNIVERSITY submitted to NRP
PHOTODEGRADATION OF AFLATOXINS M1 AND B1 IN HIGHLY TURBID LIQUID FOODS USING SHORT WAVE-LENGTH ULTRAVIOLET LIGHT
Sponsoring Institution
National Institute of Food and Agriculture
Project Status
COMPLETE
Funding Source
Reporting Frequency
Annual
Accession No.
1014558
Grant No.
(N/A)
Cumulative Award Amt.
(N/A)
Proposal No.
(N/A)
Multistate No.
(N/A)
Project Start Date
Nov 8, 2017
Project End Date
Sep 30, 2020
Grant Year
(N/A)
Program Code
[(N/A)]- (N/A)
Recipient Organization
TENNESSEE STATE UNIVERSITY
3500 JOHN A. MERRITT BLVD
NASHVILLE,TN 37209
Performing Department
Agricultural and Environmental Sciences
Non Technical Summary
Exposure to aflatoxins is known to cause both chronic and acute hepatocellular injury. While aflatoxin B1 (AFB1), aflatoxins B2 (AFB2) are found as contaminants in food derived from land farming, their metabolites (AFM1 and AFB1), occur as contaminants in food of animal origin including meat and milk. Thus, humans are at a high risk of getting exposed to different types of aflatoxins depending upon the type of foods being consumed by them. Of all the food-contaminating aflatoxins, AFB1 is usually the predominant mycotoxin and the International Agency for Research on Cancer (IARC) has classified AFB1 in the most toxic group of carcinogens (Group 1), due to the association of AFB1 exposure to liver cancer (IARC 1993). High thermal stability of mycotoxins makes them resistant to high temperatures and increase the risk of exposure in the population. In fact, several techniques have been reported as means of reducing the levels of aflatoxins. These include; heating at high temperatures, use of radiation light grinding, washing, and use of adsorbents or chemicals. This is the first study which evaluates a medium pressure and low pressure lamps source to reduce AFM1 and AFB1 and assess the cytotoxicity of treated AFM1 and AFB1 in milk. In this proof of concept based study, a laboratory photo reactor (i.e. collimated beam) using a medium pressure lamp or low pressure lamp source will be used to reduce AFB1, AFM1 in milk. This study will investigate the effect of UV irradiation on aflatoxins degradation in milk and determines the effectiveness of UV irradiation treatment of in milk against aflatoxins-induced cytotoxicity for human hepatoma cell line (HepG2).
Animal Health Component
30%
Research Effort Categories
Basic
50%
Applied
30%
Developmental
20%
Classification

Knowledge Area (KA)Subject of Investigation (SOI)Field of Science (FOS)Percent
72372992020100%
Goals / Objectives
The US food industry is now facing critical changes in response to consumer needs in terms of safety and quality of foods. The risk of contamination by mycotoxins is an important factor in food safety. Mycotoxins are toxic by-products of mold infestation, affecting one quarter of global food and feed crop output (Krska et al. 2008). Food contaminated with mycotoxins, particularly with aflatoxins, a subcategory, can cause sometimes fatal acute illness and are associated with cancer risk. AFM1 can cause DNA damage, chromosomal abnormalities, gene mutations, and cell transformation depending on the level of exposure (Michlig et al., 2016; Van Egmond, 1989). AFM1 has been designated a group 1 toxin, indicating that it is possibly carcinogenic to humans, by the International Agency for Research on Cancer (Sugiyama, Hiraokai, & Sugita-Konishi, 2008; IARC, 2012). Milk is a nutrient containing proteins, fatty acids, minerals, and vitamins, which are necessary for human health. Milk and milk products are consumed in large quantities globally, and the presence of AFM1 in milk and milk products poses a health risk. As a result of this, many experimental, clinical, and epidemiological studies have been conducted showing adverse health effects in humans and animals exposed to AFs contamination, depending on exposure (Binder, Tan, Chin, Handl, & Richard, 2007; Fung & Clark, 2004; Sherif et al., 2009). The toxicity caused by multiple mycotoxins can be classified as acute or, in the case of low dose exposure over a long period of time, chronic, leading to cancers and other irreversible effects. The prevalence of AFM1 in raw milk and milk products has been reported in many countries (WHO, 2010). UHT (ultra-high temperature) treatment of milk and liquid milk products is the application of heat to a continuously flowing product using such high temperatures for such time that renders the product commercially sterile at the time of processing. When the UHT treatment is combined with aseptic packaging, it results in a commercially sterile product. UHT treatment is normally in the range of 135-150 °C in combination with appropriate holding times necessary to achieve commercial sterility" (FAO & WHO, 2007). There have been varying reports on the effect of heat either through pasteurization or sterilization on the AFM1 in milk. For example, Studies have shown that there was no significant changes of AFM1 concentration after heat processing (Pasteurization or boiling) or Ultra-high temperature processing (UHT) technique (Galvano et al. 1996).. The above described issues of mycotoxin contamination is a nation-wide problem within USA and needs to be addressed with novel strategies and intelligent research initiatives.ObjectivesThe strategic objective of this research project is to operationally define and engrain within the consortium through disciplined implementation, a holistic Roadmap for accelerating the innovation process in irradiation research for aflatoxins detoxification. This roadmap will guide technology development for contaminant treatment (AFB1, AFM1) by integrating, in a comprehensive framework, key aspects such as customer needs, techno-economic feasibility, process modeling, technology development and validation.The specific objectives of this project are:Perform effective management of all project work and resources;Develop analytical methods for quantification of aflatoxins (AFB1, AFM1) in liquids food such as milk;Determine and verify dose delivery in liquid foods spiked with aflatoxins;Assessment of Cytokine modulation using murine macrophage cell line;Cytotoxicity and cell viability assessment;Aflatoxin induced DNA damage and DNA-adduct assessment;Develop kinetic models using MATLAB
Project Methods
Optical Property Measurements and Simulation. Optical property measurements procedure will be carried out as described by Jacques (2013); Gunter-ward et al., (2017). Light transport in highly opaque fluids is governed by radiative transport equation and does not follow Beer-Lambert law. Optical properties will be obtained using solutions of the radiative transport equation that expresses the optical properties in terms of readily measurable quantities. A state-of-the-art integrating sphere instrument will be used to collect all light scattered by the sample. The optical properties, i.e., the absorption coefficient, the scattering coefficient, and the anisotropy parameter g, will be determined by measurement of the reflectance, transmittance and collimated transmittance of thin samples. A numerical solution based on the inverse problem will be implemented to solve the radiative transport equation and deduce the optical properties of opaque fluids. This process will separate the absorption from the scattering coefficient to quantify the correct optics. In conventional absorption spectroscopy on turbid fluids, the effect of scattering is often ignored or rather simple correction schemes are adopted.UV Dose Determination. UV dose determination will carried out by developing a MATLAB based Monte Carlo program to deduce the photon fluence in a 5 ml volume in a 10ml beaker. The standard arguments for the program development will be film thickness [m], µs= reduced scattering coefficient [1/m], µa= absorption coefficient [1/m], n = refractive index, z = the depth where fluence is to be calculated, r = a vector of the radius elements where fluence is to be calculated [m], P = input power [W]. Monte Carlo convolution method will be used to quantify the fluence rate. The simulations will automatically assume irradiance to be 1W/m2. This will be adjusted to get the fluence for the correct irradiance values. The program has been described by Wang (1995). RED will be calculated as per a method described by Islam &Patras (2016); Chandra & Patras (2017).HPLC analysis of Mycotoxin Destruction. A Shimadzu HPLC system equipped with a florescence detector, (Model: RF20A) will be used for analysis of AFB1, AFM1. The method uses a 250-mm long Supelco® C18column, 4.6 mm, 5µm (Phenomenex, California, USA) as a stationary phase and a mixture of water/acetonitrile/methanol in the ratio of 60:20:20 as the mobile phase. Aflatoxins (AFB1, AFM1) will be detected using emission and excitation wavelengths of 365 nm and 440 nm respectively. Separation will be achieved at room temperature under isocratic flow mode with a flow rate of 1 ml. min-1. The range of calibrated concentration ranges between 1037.636 ± 1.24 to 255.27 ± 4.25 ng/ml, 289.05 ± 0.43 to 70.60 ± 0.33 ng/ml and 1115.99 ± 1.32 to 271.97 ± 1.32 ng/ml for AFG1, AFB2 and AFB1 respectively. The LOD and LOQ values will be calculated considering a signal-to-noise ratio of 3:1 and 5:1, respectively (Patras et al., 2017).Aflatoxins degradation analysis LC-MS/MS. The identification of AFB1, AFM1, and the respective degraded products will be carried out with an LC-MS method using a Shimadzu Prominence XR UHPLC system (Shimadzu Scientific Instruments, Columbia, MD) which included two Shimadzu LC-20ADXR pumps, a SIL-20ACXR autosampler, a CTO-20A column oven, and a Shimadzu LCMS 8030 triple quadrupole mass spectrometer. Chromatographic separation will be achieved with Phenomenex Kinetex 2.6 C18column (50 × 2.1 mm, 2.5 μm) maintained at a temperature of 40 °C. The mobile phase will consist of 0.1% formic acid in water (A) and 0.1% formic acid in acetonitrile (B). The flow rate will be 0.5 mL/min. Initially, solvent B concentration will be 5% and will increase linearly to 95% until 4.5 min. Solvent B will be returned to 5% at 4.51 min and will remain at 5% until the end of the time program at 6.0 min. The injection volume will be 5 μL. For LC-MS detection, an electrospray ionization source will be utilized in the positive ion mode with the following parameters: DL temperature, 250 °C; nebulizing gas flow, 3.00 L/min; heat block, 450 °C; and drying gas flow, 20 L/min. Control and experimental samples will be injected and data acquired in the scan mode to search for degraded products. For any potential degraded products, an LC-MS/MS product ion scan method was set up with to scan for products between 100 and 500 m/z at 15,000 amu per second with the appropriate precursor ion identified in scanning mode. Selected ion monitoring (SIM) MS methods will be developed for monitoring the following ions: AFB1 (m/z 313), AFB1-degraded products (m/z 303, m/z 311, and m/z 331), AFB1-degraded product (m/z 301), Patras et al., 2017).Cell viability assay. HepG2, CCD-18Co and HCT-116 cells will be seeded in a 24-well plate at a seeding density of 2X105cells per well. HepG2 and CCD-18Co cell will be grown in Eagle's minimum essential medium (EMEM) while HCT-116 will be grown in McCoy's 5a medium. All the cells will be supplemented with 10% fetal bovine serum (FBS) and incubated in a humidified chamber at 37°C and 5% CO2condition. Following 24 h, the cells will be serum starved overnight in a 1 mL respective media containing 1% FBS. The cells will be treated with 10 ml of the reconstituted samples for either 12 or 24 h. At the end of the exposure time the cell viability will be measured using CCK-8 reagent as per manufacturer's instruction (Dojindo Molecular Technologies, Inc., Rockville, MD). The absorbance was read at 450 nm with a reference wavelength of 650 nm in a Synergy 2 multi-mode microplate reader (BioTek, Winooski, VT).Lactate dehydrogenase (LDH) and cytokine assay. At the end of the sample exposure to the mammalian cells, the cell suspensions will be centrifuged at 400 g for 5 min and supernatant will be collected for the estimation of LDH and cytokines. For the determination of LDH, a 100 ml of supernatant will be added to 96-well plate containing 100 ml of reaction mixture (Roche Applied Sciences). The plate will be incubated for 30 min at room temperature. The reaction will be stopped using a stop solution and absorbance measure at 492 nm wave length with a reference wave length at 690 nm. Determinations of of TNF-a , IL-6, IL-10, and IL-12p40 will be all carried out by commercially available enzyme- linked immunosorbent assay (ELISA) kits obtained from R&D (Minneapolis, MN, USA).DNA-adduct ELISA assay. Cell suspension following sample exposre will be lyzed to collect DNA using DNA extraction kit (Thermo Fisher Scientific, Waltham, MA). Aflatoxin B1-DNA adducts (ring-opened and ring-closed forms) standard along with DNA collected from cell exposed with UV-treated samples will be added to 96-wells plate coated with aflatoxin B1 (AFB1)-DNA and incubated for 10 min on an orbital shaker. After incubation, 50 ml of diluted anti-AFB1-DNA antibody will be added to each well and incubated at room temperature for 1 h with constant shaking. The plate will be washed 3 times with 250 ml of 1X wash buffer and 100 ml of diluted secondary antibody-HRP will be added to each well and incubated for 1 h at room temperature. The plate will be washed 3 times with wash buffer and 100 ml of substrate solution will be added and incubated at room temperature for 2-20 min with constant shaking. The reaction will be stopped with an addition of 100 ml stop solution and absorbance will be measured at 450 nm wave length (Cell Biolabs, Inc., San Diego, CA, USA).?

Progress 11/08/17 to 09/30/20

Outputs
Target Audience:During this reporting perod, the target audience for this project is Aquafine Corporation, Trojan Technologies, Unique Food Solutions, Food Craft. Tennessee State University is working with these industrial partners to come up with novel ways to degrade mycotoxins in highly turbid liquid foods such as whole milk and almond milk. Changes/Problems: Nothing Reported What opportunities for training and professional development has the project provided?A graduate student was trained in analytical method development, dose delivery and dose validation. The student wasalso trained on complex calculations of quantum yield and optical measurements. Unfortuantely the student passed way in a tragic accident. How have the results been disseminated to communities of interest?The results were disseminated to industrial stake holer group. A fact sheet for educational purposes will be developed. A useful resource for increasing consumers knowledge in technology development and implementation. What do you plan to do during the next reporting period to accomplish the goals? Nothing Reported

Impacts
What was accomplished under these goals? Herein, we developed and engineered a novel light-based technology to degrade and detoxify AF in high risk foods. The hypothesis is that the formation of short lived reactive species initiated by UV-A photons may cause photolysis of AFB1 and AFM1 molecules, hence irradiation of aflatoxins using UV-A light at 365 nm could degrade aflatoxins via photo-oxidation (direct or indirect) without significant effect on product quality. Initially, we conducted computational fluid dyamics, artificial neural network modelling and determined the volumetric fluence rate based on the optical properties of the target fluid. Optical properties were extracted by solving the radiative transport equation using 'Inverse-Adding- Doubling'. Quantifiable UV doses were delivered to the target fluids (i.e. whole milk) in the range 0 to 720 mJ.cm-2 . Two test fluids were selected to test and validate the technology. Whole milk (WM) or almond milk (AM)samples were spiked with known concentration of AFB1 and AFM1 prior to irradiation. AF in the UV treated and control samples were quantified with reverse phase high performance liquid chromatography (HPLC) coupled with fluorescence detector. LC-MS/MS single ion monitoring was used to identify and quantify degraded product ions of AFB1 (m/z 313) and AFM1 (m/z 329). The high-resolution mass chromatogram showed two product ions of AFB1 (C17H14O7, m/z 331; C16H14O6, m/z 303) and AFM1 (C17H14O8, m/z 347; C17H10O7 m/z 327). Molecular docking studies will be conducted on selected target proteins (Acetylcholinesterase, Aldo-keto-reductase family 1 member C3 etc.) shown to interact with AFB1 and AFM1. Parent and daughter compounds werebe docked to the binding sites of the target proteins and binding energies were evaluated. Our experiments showed a degradation kinetic rate constant of AFB1 in WM as 0.0023 cm2/mJ and AFM1 as 0.0015 cm2/mJ. UV fluence at 720 mJ.cm-2 in WM reduced AFB1 and AFM1 to 82% and 86%, p<0.05. Similar results for obtained for AM. The quantum yield of AFB1 and AFM1 in WM was found to be 0.0155 and 0.0105 with an energy absorption of 5.44×10-19 J. Cytotoxicity analysis of UV-A treated samples using HepG2 liver cells showed increase in cell viability as the dose increases from 0 to 720 mJ.cm-2 and no cytotoxicity was observed at UV-A dose of 720 mJ.cm-2 . These results confirmed that the degraded AFB1 and AFM1 products in WM aresafe without any effect on cell viability. This study clearly demonstrated the efficacy of UV-A LED system in reducing AF in WM and AM below FDA recommended limits. Future study is focused on validation and development of kinetic models and evaluating the performance of this system on complex matrix such as edible oils. Based on the data obtained in this study, a flow-though UV-A system will developed.Though powerful in its proof-of-principle, the implementation of such a system in a food industry setting is challenging. Typical UV irradiation research studies utilize batch reactors (i.e., collimated beam devices); however, continuous-flow reactors are significantly more desirable for industrial food processes.

Publications

  • Type: Journal Articles Status: Published Year Published: 2020 Citation: Stanley, J., Patras, A., Pendyala, B. et al. Performance of a UV-A LED system for degradation of aflatoxins B1 and M1 in pure water: kinetics and cytotoxicity study. Sci Rep 10, 13473 (2020). https://doi.org/10.1038/s41598-020-70370-x
  • Type: Journal Articles Status: Other Year Published: 2020 Citation: Kurup, A., Patras, A., Pendyala, B. et al. Evaluation of UV-A generating LED Technology on the reduction of artificially contaminated AFB1 And AFM1 In Whole Milk: A Toxicity and Kinetic Study. Food Research International (under preparation)


Progress 10/01/18 to 09/30/19

Outputs
Target Audience:The target audiences reached by this project includes UV equipment manufacturers, regulatory agencies, food companies, academic faculty and researchers and graduate students. Changes/Problems: Nothing Reported What opportunities for training and professional development has the project provided?Two graduate students were trained on food irradiation, optical modelling, dose calculations and verification. How have the results been disseminated to communities of interest? Nothing Reported What do you plan to do during the next reporting period to accomplish the goals?Preliminary studies were conducted on milk in this reporting period. More validation studies will be conducted on whole and other milk types (3%mf, 2%. 1%). LC/MS/MS studies will conducted including cell culture studies. Milk quality will also be assessed including lipid peroxidation.

Impacts
What was accomplished under these goals? During this reporting period, the efficacy of a light emitting diode system (LED) to reduce the concentrations of aflatoxin B1, aflatoxin M1 (AFB1, AFM1) in pure water and whole milk (WM) was investigated. Irradiation experiments were conducted using a LED system operating at 365 nm (monochromatic wave-length), 3x3 configuration, operating at 40 V and 3 amp. The optical absorbance of the solutions and the irradiance of the lamp was considered in calculating the average fluence rate. Based on these factors, the UV dose was quantified as a product of average fluence rate and treatment time. Known concentrations of aflatoxins were spiked in water or WM and irradiated at UV doses ranging from (0 - 1000 mJ/cm^2). The concentration of AFB1, AFM1 was determined by HPLC with fluorescence detection. LC-MS/MS product ion scans were used to identify and semi-quantify degraded products of AFB1 and AFM1. The UV-treated degradation of AFB1 and AFM1 and the formation of the degraded products were analyzed by determining the areas of single ion monitoring (SIM) chromatographic peak areas in LC-MS experiments. LC-MS peak areas for AFB1 and AFM1 showed significant decreases with UV treatment compared to controls. Peak areas for degraded products showed an increase for each increased level of UV dose. Monitoring the formation of degraded products by LC-MS chromatographic peak areas is a qualitative approach to approximate the amount of degraded products produced. A quantitative approach would require authentic standards of the degraded products which are not readily available. AFB1, by itself is not mutagenic and it has to be bioactivated by undergoing epoxidation in the double bond in the furan ring and it is the key active site for its toxic and carcinogenic activities as the aflatoxin-DNA and the aflatoxin-protein interactions take place occur. It is envisaged that detoxification strategies should involve removal of the double bond structure and the opening of the lactone ring. From the proposed structure of the degradation products, it is evident that their structure remains almost similar to the parent compound AFB1 and only one of the two toxicological sites of AFB1 has been destroyed. It was observed that UV irradiation significantly reduced aflatoxins in pure water and WM (p<0.05). Irradiation doses of (0 - 1000 mJ/cm^2) reduced concentrations > 70% (p<0.05). Using this technique, an overall reduction of total aflatoxin content of 75 % (p<0.05) was achieved in water. We hypothesize that the formation of •OH radicals initiated by UVA light may have caused photolysis of AFB1, AFM1, molecules. Optical modelling was conducted and optical properties of WM were quantified, transmittance was calculated as 40 %/cm. Appropriate UVA doses were delivered to WM. It was observed that UV irradiation significantly reduced AFM1 in WM (p < 0.05). The reduction was observed to be 88 % at the maximum UV dose of 800 mJ/cm^2. In cell culture studies, our results demonstrated that the increase of UV dosage decreased the aflatoxins-induced cytotoxicity in HepG2 cells. The cytotoxicity test which tests for the viability of the cells in the presence of degradation products of AFB1 and AFM1 proved this presumption, but the cytotoxicity at the maximum applied dosage was found to be negligible which clearly demonstrated the efficiency of UV degradation. Therefore, our research finding suggests that UVA irradiation can be used as an effective technique for the reduction of aflatoxins.

Publications

  • Type: Journal Articles Status: Other Year Published: 2019 Citation: Performance of a novel light emitting diode (LED) system for degradation of Aflatoxins B1 and M1 in pure water: a kinetic and cytotoxicity study.Submitted Food Control (submitted)


Progress 11/08/17 to 09/30/18

Outputs
Target Audience:During this reporting perod, the target audience for this project is Aquafine Corporation, Trojan Technologies. Tennessee State University, Unique Food solutions. Changes/Problems: Nothing Reported What opportunities for training and professional development has the project provided?A graduate student was trained in analytical method development, dose delivery and dose validation. He was also trained on complex calculations of reduced scattering and absorption, including dose delivery. The student recently graduated with a Master's degree. How have the results been disseminated to communities of interest? Nothing Reported What do you plan to do during the next reporting period to accomplish the goals?Further studies on understanding the degradation mechanism of aflatoxins by UV irradiation is required. Higher doses will be deliverered to achieve 50% reduction in the concentration of AFB1. Polycromatic lamps will be used in the future to enhance the reduction ofAFB1.

Impacts
What was accomplished under these goals? Aflatoxins are fungal metabolites produced by certain strains of fungi under suitable conditions of humidity and temperature. This increases the probability of occurrence of Aflatoxins in peanut meal fed to poultry and dairy animals. In China, about 42% of dairy cow feed samples were found to contain Aflatoxin B1 from 0.05 to 3.53 µg/kg (Han et al. 2013). AFB1 contaminated meal is consumed and excreted by dairy animals. This phenomenon results in conversion of AFB1 to AFM1 in the milk being produced (Prandini et al. 2009). AFM1 requires high dry heat temperatures for degradation (about 250 °C) (Oldham and Scott 1991). To address this issue, novel pasteurization techniques are needed to degrade AFB1in milk. Task 1, 2,3: Under this reporting period, the effect of UV irradiation (low pressure lamp) on aflatoxins B1 in milk was investigated using a collimated beam system. Prior to any irradiation experiments, optical properties of milk was determined using advanced optical techniques. Absorption, scattering and refractive index of milk samples were determined. Absorption (0.573±0.030, 1/mm) and scattering (2.033±0.144, 1/mm) coefficients were mathematically extracted and ultra-violet transmittance of milk was quantified. Ultra-violet transmittance of milk was about 3.24E-01 %/cm. Based on the absorption and scattering values, known UV doses were calculated and delivered to milk samples. UV dose delivery was verified using a bacteriophage MS2 (know UV sensitvity of 20 mJ/cm^2). AFB1 was bought from Sigma-Aldrich (St. Louis, USA). 1% fat milk was bought from a local store and diluted up to 5 times with Milli-Q water. 1 mg of AFB1 was dissolved in 5 ml of methanol to make the stock solution. 200 µl of stock solution was made upto 20 ml with Milli-Q water. Serial dilutions were made from the stock solution of 2, 1, 0.5, 0.25 and 0.125 µl/ml respectively. For experimental samples, the stock solution was diluted with milk (5 fold diluted) by 200 times to yield 1 µg/ml AFB1 solution in milk and treated as discussed above. After UV treatments, solid phase extraction was carried out to remove and proteins, lipids and other small molecules which may interfere in the analysis. For solid phase extractionStrata-X 33 µm polymeric reversed phase, 200 mg/3 ml cartridges were bought from Phenomenex®, USA. The cartridges were connected to vacuum suction chamber and conditioned with 3 ml of methanol and equilibrated with 3 ml of water. 3ml of sample was loaded on the cartridges and extracted at a slow and steady rate. Later the it was washed with 3 ml of 5:95 methanol, water solution and dried at full vacuum for 10 minutes. Finally, the cartridge is eluted with 3ml of methanol and collected in HPLC grade vials for analysis. AFB1 was analyzed using reversed phase HPLC using the procedure described by Dong et al. (2010). An Agilent 1100 series instrument equipped with a quaternary pump and UV-VIS detector (Diode Array Detector, Agilent, CA) was used. The analytical column was from Phenomenex®, Luna 5u C18 150 3 4.6 mm 5 mm pore size, with a guard column equipped with Ultra cartridges (C18, 4.6 3 5mm Phenomenex®, CA). An isocratic method was used with a mixture of 60% acetonitrile and 40% water as mobile phase. The flowrate was fixed at 1.5 ml/min with an injection volume of 5 µl. This method uses UV/VIS detector at a detection wavelength of 220 nm. 6-8 min runtime was required with quick detection time. In this proof-of-concept study, the efficacy of a low-pressure lamp source to reduce the concentrations of aflatoxin B1 in milk was investigated. Irradiation experiments were conducted using a collimated beam system operating at 254 nm wave-length. The optical absorbance of the solution and the irradiance of the lamp are considered in calculating the average fluence rate. Based on these factors, the UV dose was quantified as a product of average fluence rate and treatment time. Known concentrations of aflatoxins were spiked in water and irradiated at UV doses ranging from 0, 100, 200, 300, and 400 mJ/cm^2. It was observed that UV irradiation significantly reduced aflatoxins in milk (p < 0.05). But the reduction was only 20% at the maximum UV dose of 400 mJ/cm^2. We hypothesize that the formation of ?OH radicals initiated by UV light may have caused photolysis of AFB1 in milk. The reduction of AFB1 can be enhanced using a 360 nm lamp. Therefore, our research finding suggests that UV irradiation can be used as an effective technique for the reduction of aflatoxins in milk.

Publications

  • Type: Journal Articles Status: Published Year Published: 2017 Citation: Patras, A., Chandra, S., Bansode, R. R., Vergne, M.J. (2017). Effect of UV irradiation on aflatoxins reduction: a cytotoxicity evaluation study using Human Hepatoma cell line. Mycotoxins Research, DOI: 10.1007/s12550-017-0291-0